Hiking is one of the better forms of exercise for people with migraines — it is aerobic, low-impact, mostly self-paced, and it happens outdoors, where the light and air tend to be easier on the nervous system than a fluorescent gym. The problem is that the trail also stacks several classic migraine triggers on top of each other: exertion, sun glare, heat, dehydration, altitude, and the pressure changes that come with mountain weather.

The goal of this article is not to talk you out of hiking. It is to help you hike in a way that respects how your brain responds to change, so the day outside does not turn into two days in a dark room.

Sauna and migraine have a genuinely mixed relationship, and anyone who tells you it is simply good or simply bad is overselling. For some people, regular sauna use is relaxing, improves sleep, and seems to reduce tension-driven headaches over time. For others, the heat itself is a fast and reliable migraine trigger. Both experiences are real, and which camp you fall into depends a lot on your particular triggers.

This article lays out the plausible benefits, the real risks, and a careful way to test whether sauna helps or hurts you — without provoking a bad attack to find out.

If you had to pick a single lifestyle factor with the strongest, most consistent link to migraine, sleep would be a leading candidate. Too little sleep, too much sleep, irregular timing, and poor sleep quality are all associated with more frequent and more severe attacks. For many people, the relationship runs both ways: migraines disrupt sleep, and disrupted sleep brings on migraines.

The encouraging part is that sleep is also one of the most modifiable triggers. You cannot control the weather or your genetics, but you can, with effort, control your sleep timing and environment. This article covers what the connection is and the specific habits that move the needle.

Ask a room of people with migraines what sets off their attacks, and stress will be near the top of nearly every list. The connection is one of the best-established in the field. What is less widely understood is that it is not only acute stress that triggers attacks — the release of stress matters too. The classic "let-down" migraine that arrives on the first day of vacation or the Saturday after a brutal week is a real and common pattern.

That makes stress a frustrating trigger, because you cannot eliminate it and you cannot perfectly time it. What you can do is lower your baseline stress load and smooth out the peaks and crashes. This article covers the techniques with the best support and how to actually use them.

Creatine has spent decades as a gym supplement, but it has been creeping into conversations about brain health — including migraine. The pitch is appealing and not unreasonable: if the migraine brain has an energy problem, and creatine helps cells store and recycle energy, maybe it helps. The honest answer is that the idea is mechanistically plausible and the direct evidence is thin. This piece lays out what we actually know rather than what the supplement marketing implies.

Hydrogen water has become a wellness-industry favorite, sold in pricey bottles and home machines with promises ranging from anti-aging to athletic recovery. Migraine has been swept into that list on the strength of an antioxidant argument. The science underneath is more interesting than pure snake oil but far weaker than the marketing suggests. Here is a clear-eyed look at what hydrogen water is, why anyone connects it to migraine, and how much weight the evidence can actually bear.

Most supplements marketed for migraine have thin evidence and confident marketing. Magnesium is one of the exceptions. It is cheap, widely available, well tolerated by most people, and backed by enough research that the major headache societies actually recommend it. That does not make it a cure, and it does not work for everyone — but if you are going to try one supplement for migraine prevention, magnesium is the most defensible place to start.

This guide covers which form to take, how much, how long it takes, and who is most likely to respond.

Riboflavin — vitamin B2 — is one of the quietest success stories in migraine prevention. It costs a few cents a day, has almost no side effects beyond turning your urine a vivid yellow, and has enough evidence behind it that headache specialists routinely suggest it. It will not work for everyone, but the risk-to-reward ratio is about as favorable as anything you will find.

This piece covers how it works, the dose that has actually been studied, and why patience is the whole game.

Sound healing covers a wide range of practices — singing bowls, tuning forks held to the body, gongs, and vibroacoustic tables that transmit low-frequency vibration through your back. It is increasingly offered for migraine and chronic pain. For a condition where many sufferers are acutely sound-sensitive, the idea of using sound as therapy is either intriguing or alarming depending on your last attack. This piece separates what is plausible from what is marketing, and offers a sensible way to experiment.

The instinct to put something cold on your head during a migraine is older than the migraine literature. Cold therapy turns up in medical writing going back to antiquity, and modern wearable cold caps are a thriving small industry. Warm compresses get used too — usually for the neck and shoulders when migraine and tension overlap. Both work for some people, neither works for everyone, and using the wrong one can sometimes make an attack worse.

This piece sorts out when cold helps, when heat helps, and how to combine them sensibly.

Ask anyone with chronic migraine to list their triggers and dehydration almost always shows up in the top five. The connection is real — but the advice that follows is usually too generic to be useful. "Drink more water" leaves out the parts that matter: how much, when in the day, with what electrolytes, and whether it actually does anything for an attack already in progress.

This piece covers what the research shows about hydration and migraine, what to do during a typical day, and how to use fluids during an attack.

Light sensitivity — photophobia — is one of the most consistent migraine symptoms. During attacks, ordinary indoor lighting can feel like floodlights, and even between attacks many people with migraine have a baseline level of glare and screen discomfort that healthy controls do not. That experience is what drives the migraine-glasses market: FL-41 rose-tinted lenses, blue-light blockers, polarized sunglasses, and various proprietary tints all claim to help.

Some of those claims are backed by real research. Others are not. This piece sorts the evidence so you can spend money on lenses that actually do something.

Migraine does not happen in a vacuum. It happens at a particular desk under fluorescent lights, on a particular commute, after a particular night of poor sleep, in a particular kitchen where breakfast got skipped again. Occupational therapy is the discipline that takes those daily-life mechanics seriously. For migraine, an OT works on the routines, environments, and demands that quietly raise your attack frequency — and on the practical strategies that let you function during an attack instead of losing the whole day.

This piece covers what occupational therapy for migraine looks like, where it fits next to medication and other treatments, and how to tell whether it would help you.

Many people who get migraines also have a neck that has been quietly making things worse for years. Tight upper trapezius, weak deep cervical flexors, restricted upper cervical joints, and a forward head posture all feed into the trigeminocervical complex, the brainstem hub where neck input and head pain converge. Physical therapy will not cure migraine, but for the meaningful subset of patients whose attacks have a cervical contributor, a good PT program can lower frequency and intensity enough to notice.

This piece covers what physical therapy for migraine actually involves, what the evidence supports, and how to tell whether it is likely to help you.

For decades, migraine prevention relied on drugs borrowed from other conditions — beta blockers from cardiology, anticonvulsants from neurology, antidepressants from psychiatry. They worked, sometimes, with side effects that often outweighed the benefit. The CGRP monoclonal antibodies were the first class of drugs designed from the ground up specifically to prevent migraine, and they have changed what the preventive landscape looks like.

This piece walks through what CGRP is, what the antibodies do, and how to think about this class in practice.

Craniosacral therapy comes up regularly when migraine patients are looking for non-drug options. It is gentle, widely available, and almost universally well-tolerated, which makes it appealing. The harder question is whether it actually works — and the answer is more nuanced than the marketing on either side of the debate suggests.

This piece walks through what craniosacral therapy is, what the evidence shows, and how to think about it in a migraine plan.

Naproxen and meloxicam both turn up in headache treatment, and both are NSAIDs, but they are working in different ways and earning their place in different parts of a migraine plan. Naproxen is the workhorse for acute migraine attacks. Meloxicam is more of a preventive-leaning, once-daily NSAID that occasionally gets pulled into migraine treatment when a steadier baseline is needed. Mixing them up — or assuming one can simply replace the other — usually leads to a less effective plan.

This piece walks through how they differ, where each one earns its place, and what the trade-offs look like.

Most migraine attacks are treated, at least initially, with over-the-counter medication. That is true even for people who later move to prescription options — most of them are still relying on an OTC drug for milder attacks. Knowing which OTC drugs work, how to combine them, and where they stop being enough is the foundation of an acute migraine plan.

This guide walks through the OTC options, what each one does well, and how to think about them in practice.

Pain reprocessing therapy is a structured, brain-focused approach to chronic pain that has accumulated enough evidence to be taken seriously — particularly for chronic back pain, where the first randomized trial showed striking results. The question for migraine patients is whether the same framework applies to chronic migraine and what it would look like in practice.

This piece walks through what PRT is, where the evidence sits, and how to think about it for migraine specifically.

Naproxen is the long-acting NSAID that most often shows up in migraine treatment plans, both as a stand-alone abortive for mild-to-moderate attacks and as the NSAID half of a combination with a triptan. It is one of the cheaper and better-tolerated members of its drug class, and the 12-hour half-life is a real advantage for migraines that drag on or rebound late in the day.

This piece covers the dosing that actually works for migraine, how naproxen compares with other NSAIDs, when to pair it with a triptan, and the safety points that matter.